VARICES ESOFAGICAS PDF

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two receding years. CiteScore measures average citations received per document published. Read more. SRJ is a prestige metric based on the idea that not all citations are the same. SJR uses a similar algorithm as the Google page rank; it provides a quantitative and qualitative measure of the journal's impact. SNIP measures contextual citation impact by wighting citations based on the total number of citations in a subject field.

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The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two receding years.

CiteScore measures average citations received per document published. Read more. SRJ is a prestige metric based on the idea that not all citations are the same. SJR uses a similar algorithm as the Google page rank; it provides a quantitative and qualitative measure of the journal's impact. SNIP measures contextual citation impact by wighting citations based on the total number of citations in a subject field.

ISSN: Previous article Next article. Issue 8. Pages January Download PDF. Corresponding author. Unitat de Sagnants. Servei de Patologia Digestiva. Hospital de la Santa Creu i Sant Pau. Antoni M. Claret, This item has received. Article information. Full text is only aviable in PDF. D'Amico, L. Pagliaro, J. The treatment of portal hypertension: a meta-analytic review. Hepatology, 22 , pp. De Franchis. Updating consensus in portal hypertension: report of the Baveno III consensus workshop on definitions, methodology and therapeutic strategies in portal hypertension.

J Hepatol, 33 , pp. Bosch, J. Feu, A. Luca, M. Pizcueta, et al. New approaches in the pharmacologic treatment of portal hypertension. J Hepatol, 17 , pp. Polio, R. Hemodinamic factors involved in the development and rupture of esophageal varices: a pathophysiologic approach to treatment.

Semin Liver Dis, 6 , pp. Groszmann, R. Fisher, H. Conn, C. Atterbury, M. Portal pressure, presence of gastroesophageal varices and variceal bleeding. Hepatology, 5 , pp. Bhatal, H. Reduction of the increased portal vascular resistance of the isolated perfused cirrhotic rat liver by vasodilators.

J Hepatol, 1 , pp. Wiest, R. The paradox of nitric oxide in cirrhosis and portal hypertension: too much, not enough. Hepatology, 35 , pp. Complications of cirrhosis. Portal hypertension. J Hepatol, 32 , pp. Luca, P. Effects of low-sodium diet and spironolactone on portal pressure in patients with compensated cirrhosis.

Hepatology, 19 , pp. Castells, A. Luca, R. Hermida, F. Rivera, et al. Circadian variations of portal pressure and variceal hemorrhage in patients with cirrhosis. Golombek, P. De Las Heras, L. Viola, S. Diurnal fluctuations of portal and systemic hemodynamic parameters in patients with cirrhosis. Hepatology, 20 , pp. McCormick, R. Dick, M. Graffeo, D. Wagstaff, A. Medden, N. McIntyre, et al. The effect of non-protein liquid meals on the hepatic venous pressure gradient in patients with cirrhosis.

J Hepatol, 11 , pp. O'Brien, M. Keogan, S. Patchett, P. McCormick, N. Afdhal, J. Postprandial changes in portal haemodynamics in patients with cirrhosis.

Gut, 33 , pp. Silva, F. Bresky, C. Backhouse, M. Palma, M. Ruiz, et al. Splanchnic and systemic hemodynamics in early abstinence and after ethanol administration in non-cirrhotic alcoholic patients.

J Hepatol, 20 , pp. Luca, F. Feu, J. Bosch, et al. Ethanol consumption worsens hepatic hemodynamics in patients with alcoholic cirrhosis and portal hypertension. Hepatology, 18 , pp. Santos, J. Luca, J. Roca, R. Physical exercise increases portal pressure in patients with cirrhosis and portal hypertension. Gastroenterology, , pp. McCormick, S. Jenkins, N. McIntyre, A.

1001 MANEIRAS DE PREMIAR SEUS COLABORADORES PDF

Várices esofágicas

ISSN: Descargar PDF. Autor para correspondencia. Unitat de Sagnants.

THIRUKURAL KATHAIGAL IN TAMIL PDF

2001, Número 4

Hemorragia digestiva alta. Portosystemic collateral formation, particularly at the gastroesophageal junction, is a most serious consequence of portal hypertension. Increased portal pressure is the most significant force underlying gastroesophageal variceal formation, to which end portal pressure estimated from the hepatic venous pressure gradient must reach at least 10 mmHg. Subsequently, splanchnic hyperemia also contributes to variceal development. Portoystemic collaterals result from repermeabilization of pre-extant vessels, vascular remodeling, and angiogenesis. The goal of pre-primary prophylaxis is preventing or delaying the formation of gastroesophageal varices. In experimental models of portal hypertension, early administration of splanchnic vasoconstrictors such as beta-blockers, nitric oxide synthesis inhibitors, or antiangiogenic substances inhibits portosystemic collateral formation.

DOCUCENTRE-II C4300 PDF

Pathophysiology and treatment of variceal hemorrhage. Mayo Clin Proc ; The treatment of hypertension portal: A meta-analytic review. Hepatology ; 22 1 ; Grace ND. Diagnosis and treatment of gastrointestinal bleeding secondary to portal hypertension. Hepatology ; 22 6 :

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